oral cancer-ADENOID CYSTIC CARCINOMA

Dec 9th 2010…. I got news you have malig tumor a vary tumor. I have Adenoid cysytic the small salivary glands. It had taken over most my , the floor mouth and my entire jaw . I had surgery to remove the bulk tumor including 60% my tongue ans we chose to leave the entire jaw bone Gods hands as It would severly disform me and I leave me without a face and on a trach for life and feeding tube for life and I did not want that for my sons nor I so I have Faith and trust God for a miracle for me and all other patients out there…God Bless us all

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Rzayev RM Ossifying fibroma of the nasal cavity-Craniofacial resection.

A 45-year-old man presented with an cavity with involving the maxillary, ethmoid sphenoid sinuses, which also penetrated intracranial. The tumor was successfully excised applying operation-anterior craniofacial

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Alternative Cancer Treatment for Squamous Cell Carcinoma & Thyroid Cancer

survivor Paul LeFebvre speaks about results obtained after receiving therapies treatment at An Oasis under the care , . www.AnOasisOfHealing.com

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Skin Cancer Squamous Cell Carcinoma of the Arms and Legs

Dr. Shane Chapman, discusses Cancer Carcinoma . See more at www.dermnet.com PLEASE RATE AND COMMENT!!! Marjolin’s ulcer is a term that refers to malignant changes occurring in ulcers and wounds the , sinuses and previous burns. Most lesions are reported in burns. The majority of these lesions are found on the extremities. Development times for burn scar carcinomas of more than 30 years have been noted. Different cultures appear to have markedly different susceptibilities to Marjolin’s ulcer. Japan, Northern India, and China report high incidences of burn-scar carcinoma. SCCs that occur at sites of chronic inflammation are more aggressive than those that develop from actinic keratoses or Bowen’s disease. Their appearance is masked by inflamed hypertrophic tissue. The overall metastatic rate is greater than 40%. The incidence of regional lymph node involvement from burn-scar carcinoma is approximately 35%. The 5-year survival rate for lower extremity lesions is approximately 30%. Because of the focal nature of malignant change in burn scars, excisional biopsy should be performed. Punch biopsies may be negative. Wide local excision has proven unreliable for grade II and grade III disease; amputation and prophylactic node irradiation is recommended. Wide local excision is reserved only for very small lesions that can be radically excised or for grade I lesions.

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CCSVI: The Liberation Therapy of Dr Zamboni

Treatment CCSVI is a condition called “ Insufficiency” was discovered by Paolo , a vascular surgeon at the University of Ferrara Italy. It refers a narrowing or blockage of the primary veins draining blood from the the heart. These include the jugular veins, veins along the spinal column and the azygos vein the upper chest. This narrowing restricts the normal outflow of blood from the brain. As a result, the blood often “refluxes” that is: it flows backwards into the brain. Some think the resulting flow and building pressure pushes blood into the tissue around vessels the brain, resulting toxic iron deposits that some believe may trigger inflammation, injury to brain tissue and cell death. Dr. has used Doppler ultrasound to scan the heads and necks of over 500 patients and found the blocked, narrowed and sometimes missing veins of CCSVI in almost 100 per cent of them. These problems were found only in patients, not in healthy people nor in those with other neurological conditions. Doctors in the United States and Poland have also scanned a small number of patients and found they too have CCSVI. Dr. Michael Dake at Stanford University in California has also been scanning patients and finding a striking similarity to Zamboni’s findings. The Liberation Treatment is still an experimental treatment and is not widely offered except as part of some studies underway. In Italy, the

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CCSVI & MS: 60 day liberation follow up. “Flat Earth” Neuros, ETOH abuse and MS.

Hello ALL. Still feel cured . was really cramping my style and I have felt great for quite awhile. :) This video is an update and talking about how good I feel. I address the resistance many neurologists have for CCSVI and why it makes no sense to be skeptical of CCSVI when it is showing much greater efficacy than so-called “ modifying drugs” which Hubbard points out that DMD’s could only be effective because of “placebo effect”. NICE STRATEGY! Use the skeptic’s argument against them. Thiamine (Vitamin B1) deficiency is seen abuse. Alcoholics have similar brain damage on MRI to MS patients as well as B1 deficiency. I also have also uncovered another comorbidity that could affect the course of MS. Chronic abuse Alcoholism Binge drinking. Daily consumption of even small amounts of could be catastrophic for MS patients. Why PWMS should not drink : : www.encyclopedia.com How chronic abuse influences demyelination LINK : archneur.ama-assn.org So am I saying people with MS are alcoholics? NO. I am saying that studying the brains of alcoholics has revealed similar damage to the brain and nerves one sees in . Think of it this way. MS patients already have brain/nerve damage happening. DO NOT DRINK IF YOU ARE AN MS’er. It could change the course of your disease for the worse. NOT DRINKING could change the course of your MS for the better,. I felt compelled to address this in this

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